TRANSPLANTATION PROCEEDINGS, vol.49, no.2, pp.281-287, 2017 (SCI-Expanded)
Background. Chronic allograft dysfunction (CAD) is the most important clinical problem in solid organ transplantation. Interstitial fibrosis and tubular atrophy contribute to long-term renal allograft failure. Urinary type III prodollagen N-terminal propeptide (PIIINP), has been shown to associate fibrotic processes.