P. Revy Et Al. , "Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the hyper-IgM syndrome (HIGM2)," CELL , vol.102, no.5, pp.565-575, 2000
Revy, P. Et Al. 2000. Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the hyper-IgM syndrome (HIGM2). CELL , vol.102, no.5 , 565-575.
Revy, P., Muto, T., Levy, Y., Geissmann, F., Plebani, A., Sanal, O., ... Catalan, N.(2000). Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the hyper-IgM syndrome (HIGM2). CELL , vol.102, no.5, 565-575.
Revy, P Et Al. "Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the hyper-IgM syndrome (HIGM2)," CELL , vol.102, no.5, 565-575, 2000
Revy, P Et Al. "Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the hyper-IgM syndrome (HIGM2)." CELL , vol.102, no.5, pp.565-575, 2000
Revy, P. Et Al. (2000) . "Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the hyper-IgM syndrome (HIGM2)." CELL , vol.102, no.5, pp.565-575.
@article{article, author={P Revy Et Al. }, title={Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the hyper-IgM syndrome (HIGM2)}, journal={CELL}, year=2000, pages={565-575} }