Raimondo, A. Et Al. 2014. Phenotypic severity of homozygous GCK mutations causing neonatal or childhood-onset diabetes is primarily mediated through effects on protein stability. HUMAN MOLECULAR GENETICS , vol.23, no.24 , 6432-6440.

Raimondo, A., Chakera, A. J., Thomsen, S. K., Colclough, K., Barrett, A., De Franco, E., ... Chatelas, A.(2014). Phenotypic severity of homozygous GCK mutations causing neonatal or childhood-onset diabetes is primarily mediated through effects on protein stability. HUMAN MOLECULAR GENETICS , vol.23, no.24, 6432-6440.

Raimondo, Anne Et Al. "Phenotypic severity of homozygous GCK mutations causing neonatal or childhood-onset diabetes is primarily mediated through effects on protein stability," HUMAN MOLECULAR GENETICS , vol.23, no.24, 6432-6440, 2014

Raimondo, Anne Et Al. "Phenotypic severity of homozygous GCK mutations causing neonatal or childhood-onset diabetes is primarily mediated through effects on protein stability." HUMAN MOLECULAR GENETICS , vol.23, no.24, pp.6432-6440, 2014

Raimondo, A. Et Al. (2014) . "Phenotypic severity of homozygous GCK mutations causing neonatal or childhood-onset diabetes is primarily mediated through effects on protein stability." HUMAN MOLECULAR GENETICS , vol.23, no.24, pp.6432-6440.

@article{article, author={Anne Raimondo Et Al. }, title={Phenotypic severity of homozygous GCK mutations causing neonatal or childhood-onset diabetes is primarily mediated through effects on protein stability}, journal={HUMAN MOLECULAR GENETICS}, year=2014, pages={6432-6440} }